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These findings underscore their potential as therapeutic agents by modulating UFMylation, offering new strategies for targeting UFMylation-dependent pathways in glioblastoma and potentially other diseases associated with dysregulated protein degradation and ER stress responses

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Brain Dev 25:377382 Araghi-Niknam M, Fatemi SH (2003) Levels of Bcl-2 and P53 are altered in superior frontal and cerebellar cortices of autistic subjects

1.5 g Other Ingredients: cellulose (capsule), vegetable stearate