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[3] The scientific interest in survodutide sits at the intersection of two of the largest unmet needs in cardiometabolic medicine: obesity and metabolic dysfunction-associated steatohepatitis (MASH, formerly called NASH)

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Enzymatically nonfunctional SsCm1 conservatively targets chloroplast RNA editing factors MORF2s SsCm1 inhibits SA biosynthesis reminiscent of Cmu1 of Ustilago maydis which inhibits SA biosynthesis by interacting with the host Cm 39