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Experimental modulation of endothelial survival, angiogenic signaling and metabolic state reveals that intact tyrosine kinase receptor (Tie2) signaling, glycolytic competence of peritubular capillary endothelial cells, and sustained VEGF-A-vascular endothelial growth factor receptor 2 (VEGFR2) communication from injured TECs are all required to maintain endothelial identity, prevent EndMT and limit microvascular rarefaction [121]

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388 In parallel with its affecting metabolic pathways, hypoglycemia also plays a role in regulating liver repair and regeneration

[7] Adenoviral vectors carrying synthetic thymulin genes achieved longer transgene expression in rat brain regions compared to other reporter genes