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93 Similarly, a truncation mutation (R3047X) located near Cys2991 in ATM results in insensitivity to oxidative stress

In the Second Opinion Physician model, impaired disposal of homocysteine, adenosine and related products may help maintain SAH accumulation

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COPZ1 knockdown in glioblastoma cell lines (U87MG, U251, and P3) induces ferritinophagy, upregulates NCOA4, and activates autophagy-related protein ATG7, leading to excessive Fe 2+ release, ROS accumulation, and ferroptotic cell death [36]