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Description

Therefore, targeting GLS to block GLN degradation is a promising therapeutic strategy

D., Ramachandran, A

Oxidative stress in chronic kidney diseases is characterized by several distinct pathways: oxidative stress mediated by ROS ( OH), nitrosative stress mediated by RNS (ONOO , NO 2 ), chlorinated stress mediated by chlorinated species (HOCl), and carbonyl stress (Advanced glycation end products, AGEs) (Ling and Kuo 2018)

This review integrates mechanistic insights with translational advances, outlining how dysregulation of chaperones, autophagy-mitophagy, the ubiquitinproteasome system, and ER stress pathways drive human diseases, while highlighting emerging therapeutic platforms, from pharmacological chaperones and autophagy modulators to targeted protein degradation technologies, CRISPR screens, spatial biology, and AI-guided drug discovery