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Neuroinflammation Neuroinflammation is increasingly recognized as a key factor interacting with neurobiological correlates of the neuropsychiatric long COVID syndrome, such as brain serotonin depletion [111], hypothalamicpituitaryadrenal (HPA) axis dysregulation [57], and impaired hippocampal neurogenesis and plasticity [13, 61]

This process occurs through the direct binding of RSL3 to USP11, resulting in its inactivation and subsequent induction of ubiquitination and degradation of NRF2 protein in these cells [122]

Tumor hypoxia and radiotherapy: a major driver of resistance even for novel radiotherapy modalities

Sung, S.-Y