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In addition to the response to infection, tissue damage itself can initiate inflammation, through the release of damage-associated molecules including arachadonic acid metabolites, free extracellular matrix molecules, nucleotides and others (Van Crombruggen et al., 2013)

The loss of protection during menopause suggests that E1, the predominant postmenopausal oestrogen, exerts limited metabolic effects compared with E2

IGF1R-6 integrin-S100A4 network governs the organ-specific metastasis of chemoresistant epithelial ovarian cancer cells

Semaglutide is currently approved only as a weekly injection under the skin or as an oral tablet