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In addition, in cultured human tubular cells, cisplatin reduced SIRT3, resulting in mitochondrial fragmentation, while restoration of SIRT3 with ALC (200 or 500 mg/kg b.w., i.p .) improved cisplatin-induced mitochondrial dysfunction [102]

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Conversely, in selenium-replete populations, supraphysiologic dosing yields inconsistent outcomes and has not reliably demonstrated cardioprotective benefit

There is a clear need for more accurate and early diagnosis of people who are on the trajectory to develop AD so clinical trials can be conducted in individuals where the course of the disease could be modified or reversed