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However, topical application of PTH 134 may lead to overactivation of osteoclasts, potentially resulting in compromised bone healing outcomes

In a rat model of cerebral ischemia-induced neurological impairment, the co-administration of tissue plasminogen activator (tPA) with nanoliposomal Fasudil-Lip showed better neuroprotection, and prolonged therapeutic time window (TTW) and improved drug targeting (Fukuta et al., 2017)

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To sum up, the TL-LNP significantly reduced the cytotoxicity of the formulation and enhanced the transfection efficiency by decreasing the intracellular oxidative stress