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Systemic Interaction of Genetic and Modifiable Factors While the genetic mutation is the root cause, the overall clinical picture of thalassemia is shaped by the interaction between inherited defects and modifiable factors such as metabolism, nutrition, and oxidative stress

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Therefore, targeting Flk1 signaling in the RPE may prevent the development of neovascular AMDlike pathologies, thereby providing a novel therapeutic rationale for the prevention of neovascular AMD