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Asian Pac J Trop Med

[DOI] [PubMed] [Google Scholar] 72.Ju J., Li X.-M., Zhao X.-M., Li F.-H., Wang S.-C., Wang K., Li R.-F., Zhou L.-Y., Liang L., Wang Y., et al

This process can be antagonized by methylation inhibitors silencing DNMT3a and SEPT9 overexpression, thereby reducing liver fibrosis, including upregulation of apoptosis-related proteins in myofibroblasts, and reduction of TGF-1-induced expression of -SMA and COL1A1

As the primary organ responsible for steroid metabolism, the liver is particularly susceptible to the toxic effects of 17--alkylated oral anabolicandrogenic steroids, such as stanozolol and methyltestosterone ( Mechanistic studies suggest that anabolicandrogenic steroids impair mitochondrial -oxidation and deplete intracellular glutathione stores, resulting in lipid peroxidation and activation of apoptotic signalling pathways