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Alanine mutations of E124 3.33 and R283 6.55 nearly abolished the binding of ghrelin-A2 and the ghrelin-induced receptor activity, indicating the critical role of the salt bridge in maintaining the integrity of the bifurcated binding pockets, and thus affecting ghrelin binding and receptor activation (Supplementary Figs

Ferroptosis-inducing compounds exert their effect primarily through the direct inhibition of GPX4, a key antioxidant enzyme responsible for reducing lipid hydroperoxides to non-toxic lipid alcohols