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Besides, Chloroquine blocks virus infection by increasing endosomal pH required for virus/cell fusion, as well as interfering with the glycosylation of cellular receptors of SARS-CoV

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IR and IGF-1R display approximately 50% sequence homology (Ullrich et al., 1986)

Additionally, the immunoregulatory function of GABA participates in comorbidity mechanisms, and its deficiency leads to elevated pro-inflammatory cytokine levels in the intestinal mucosa of comorbid patients, forming a vicious cycle of inflammation-neurotransmitter dysregulation (Seifi et al., 2018)