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doi: 10.1152/ajpgi.00081.2014 261 LichtenLALiuzziJPCousinsRJ

BPC-157 works through several biological mechanisms that promote healing and reduce inflammation at the tissue level

Drug-responsive autism phenotypes in the 16p11.2 deletion mouse model: A central role for gene-environment interactions

Elevated ROS exacerbate endothelial damage, stimulate VSMC proliferation, and drive vascular remodeling, thereby raising peripheral resistance and blood pressure [245]