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These activities included increased body mass, growth of visceral organs, and possibly improved feed utilization efficiency when models were continuously exposed to the agent

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[] There is controversy about the liver to the skeletal muscle crosstalk in patients with comorbidity of NAFLD and sarcopenia, as that NAFLD and sarcopenia share many complex interplaying mechanisms, including insulin resistance and myostatin and adiponectin homeostasis, which make it difficult to determine the actual cause, therefore, identifying whether sarcopenia is a cause, consequence, both, or neither of fatty liver is challenging