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J Clin Oncol (2001) 19(2):51924

Clinically, many late-onset MADD patients respond dramatically to high-dose riboflavin (100400 mg/day), confirming residual enzyme function amenable to cofactor restorationclassifying MADD as a prototype FAD-responsive metabolic myopathy (70)

For example, muscle overuse-induced cysteine oxidation and its intermediate cysteine sulfinic acid oxidation result in a reduced level of taurine and glutathione synthesis (GSH), ultimately compromising the taurine-induced benefits in the muscle, such as influencing GSH level and glutathionylation (Qin et al., 2013)

Bae et al., 2 O 2 leads to the oxidative stress-induced aggregation of -syn by crosslinking -syn tyrosine residues through dityrosine bonding (Hashimoto et al., 1999