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Moreover, it has been shown that chronic ammonium exposure in cultures of the granule neurons of the cerebellum induces the redistribution of protein kinase C (PKC) isoforms in the membrane and cytosol, thus causing cell dysfunction (Giordano et al., 2005)

The protein level of significantly increased p16 was confirmed in keloid lesions and fibroblasts based on western blot analysis ( p = 0.0007, p = 0.03

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While the Akt/eNOS, NOX and Nrf2 pathways have been widely studied in the literature, it has been shown that phenolic compounds can modulate various cellular functions indirectly related to the redox balance regulation, such as inflammation or endothelial dysfunction (Reference Ruskovska, Maksimova and Milenkovic71)