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High levels of miR-124 in serum inhibit the expression of Fpn, leading to iron accumulation and ferroptosis in cells ( FIGURE 1 TABLE 1 The regulatory mechanism of ferroptosis

The disrupted synaptic function in ASD relates to higher-level phenotypic changes as observed in other neurologic disorders and may result in altered sensory processing, cognitive deficits, hyperactivity, and seizures by affecting the balance between excitatory and inhibitory neurotransmission [4]

6,7 Although the term ferroptosis was coined in 2012 after screenings for small-molecule compounds capable of inhibiting the growth of RAS-mutant cancer cells 8 (Fig

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