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Nanomaterial-mediated modulation of long-range neural circuits and projection pathways for epilepsy therapy Excessive neuroinflammation is a core pathological feature of epilepsy, driven in part by the aberrant activation of microglia and astrocytes

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However, many factors, including their low stability, short half-life, high immunogenicity, tissue targeting, cellular uptake, and endosomal escape, hamper the delivery and clinical application of NADs

A strong response does not always mean the therapy is wrong, but it may mean the pace is wrong