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During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes

[DOI] [PMC free article] [PubMed] [Google Scholar] 346.Wu Y, et al

Building on previous findings, the current research aimed to assess the protective roles of BCA, CoQ10, and PHL against oxidative stress, nephritic apoptosis, and molecular disruptions caused by simultaneous chromium and arsenic exposure in Swiss albino mice

To validate the phosphorylation relationship between PEPR1 and BAK1 in vivo , we co-expressed PEPR1-Km-GFP with BAK1-mCherry in the Nicotiana benthamiana leaf epidermis with or without Pep1 (500 nM) treatment for 15 min, and performed MS-based phospho-analysis (Supplemental Dataset 2)