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The fact that folinic acid, even at high dose, did not change metabolic markers of methylation and/or glutathione metabolism unless it was combined with mB12 is an important finding as it has direct implication for treatment of inter-related but distinct metabolic disorders associated with ASD

Homocysteine thiolactone: metabolic origin and protein homocysteinylation in humans

Meanwhile, the vector backbone of pTH19Kr plasmid was produced by PCR with primers (5-caagcttggcgtaatcatgg and 5-taatgctctgccagtgttac)

Conclusion: Lack of VMP1 or TMEM41B leads to impaired VLDL secretion and steatosis that can be reversed via selective clonal hepatocyte expansion