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One study found that MS patients had higher levels of Akkermansia and Acinetobacter and lower Parabacteroides compared to controls, and when these microbial communities were transferred to germ-free mice, the mice developed more severe EAE (the mouse model of MS) [184]

It has also been shown to enhance muscle repair and the cellular remodelling that accompanies new muscle tissue

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The question researchers keep returning to is whether restoring GH pulsatility in aged subjects reverses measurable functional decline in muscle, bone, and metabolic parameters, or whether the somatopause itself reflects a broader neuroendocrine recalibration that GH restoration cant fully address