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The oxidation of fatty acids is affected as a result of the metabolism of fructose by fructokinase C, thus the result of the consumption of adenosin triphosphate (ATP), the production of uric acid and the nucleotide exchange, and finally, concomitant mitochondrial dysfunction [55]

This combination creates a potent detoxification and anti-aging protocol that supports immune function, collagen synthesis, and cellular repair processes

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Moreover, it has been demonstrated that LCKD enhances cardiac tolerance to global ischemia (Al-Zaid et al., Med30 zg mutation (Krebs et al., Med30 zg mutation causes a progressive and selective decline in the transcription of genes necessary for oxidative phosphorylation and mitochondrial integrity, eventually leading to cardiac failure