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Oxidative stress is a hallmark of ARDS, with elevated ROS levels initiating and propagating lipid peroxidation ( Inflammation also amplifies ferroptosis through cytokine-mediated pathways

In human subjects with T2DM, treatment was associated with improved HbA1c levels and lipid profiles, suggesting that resolving systemic inflammation is a viable strategy for metabolic correction

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2 There is also a much-discussed link to the Wnt/-catenin pathway, which is a genuine master regulator of the hair cycle: active -catenin signaling in the dermal papilla drives follicles into anagen and enhances their hair-inducing capacity, while reduced signaling is associated with regression and miniaturization