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626 The absence of ALOX5 expression eliminates HTTQ94-induced ferroptosis under oxidative stress conditions, suggesting that ALOX5 is essential for ferroptosis mediated by mutant huntingtin and could be a promising new target for HD treatment

Gene Ontology (GO) enrichment analysis revealed that NB4-specofic proteins are primarily associated with enzymatic functions and enriched in exosome and myelin sheath, whereas HL60-specific proteins exhibit RNA-binding properties without specific localization [30] (Fig

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Bruno Bastide, Lille University of Science and Technology, France Updates Copyright 2019 Andrich, Melbouci, Ou, Auclair, Mercier, Grenier, Lira, Barreiro, Danialou, Comtois, Lavoie and St-Pierre