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GFP fluorescence levels were significantly increased in worms fed mutant diets compared to the control diet, indicating UPRmt activation (Fig

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For example, PTEN mutations, which occur downstream of EGFR signaling, have been identified as a resistance factor in anti-EGFR therapy for GB, highlighting the complexity of these pathways and the challenges in developing effective targeted treatments ( Figure 1 further illustrates the intricate and overlapping signaling cascades within the RTK pathway, emphasizing the complexity involved in the context of the various targeted therapeutics discussed above

Keap1 binds to Cul3 via its BTB domain, forming the Keap1-Cul3-RBX1 E3 ligase complex, which plays a crucial role in the ubiquitination of NRF2 [88, 89]