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Abnormalities in selenium metabolism and skeletal development diseases Selenium deficiency is regarded as one of the initiating factors of KBD, which is an endemic osteoarthropathy caused by the premature closure of epiphyseal plate and the impaired skeletal development

Pathological astrocytes are distinguished by the expression of a specific marker WD Repeat Domain 49 (WDR49), while immunohistochemistry demonstrated that WDR49-positive astrocytes form clusters that are scattered randomly throughout the cortex of patients with FTD 225,226

Physiological roles : transport of nitrogen in a non-toxic form and of the gluconeogenic substrate pyruvate to the liver, thereby shifting the metabolic burden associated with nitrogen disposal and glucose regeneration to the liver

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