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Additionally, we summarize the progress of ongoing clinical trials targeting the 1C network, highlighting the potential to exploit metabolic vulnerabilities for cancer therapy

Loss of sirt1 promotes prostatic intraepithelial neoplasia, reduces mitophagy, and delays park2 translocation to mitochondria

Increasing efforts have been endeavored to identify potential inhibitors against gasdermins, which are key executors in pyroptosis and are expected to be promising therapeutic targets for diverse diseases including but not limited to septic shock, cancer, and tissue damage

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