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Hence, phenotypic alterations observed when the function of the GPx4 5UTR is compromised either by siRNA targeted at these sequences (Borchert et al., 2006) or by manipulating the expression of RNA-binding proteins such as Grsf1(Ufer et al., 2008) that bind these sequences reflect the importance of this regulatory mechanism for GPx4 expression

Similar content being viewed by others Introduction The innate immune recognition of potential pathogenic microorganisms comprises the recognition of microbial-associated molecular patterns (MAMPs) and endogenous signals or damage-associated molecular patterns (DAMPs), which derive from cellular damage caused by infections