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Therefore, here we used hyperphenylalaninemic (Hph-1) mice, deficient in GTPCH with the heterozygotes and homozygote animals producing different BH 4 levels to demonstrate the relationship between BH 4 and NOS output, and sepiapterin supplementation to investigate the impact of an improved BH 4 supply in the ischaemic phase of the murine OIR model

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However, the overall adipocyte area was dramatically reduced in a dose-dependent manner after the S

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