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The rationale is based on the known biochemistry of fatty acid oxidation and the established (though modest) effects of levocarnitine supplementation on body composition in separate studies

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The results showed that TRIM36 hypermethylation was found in 90.0% (27/30) of the NSCLC, indicating that BaP induced hypermethylation of TRIM36, then promoted lung carcinogenesis [55]