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The low-desire studies reported no treatment-related serious adverse events

Upon recruitment, the membrane adhesion proteins such as intercellular adhesion molecule-1 on endothelial cells are up-regulated, and the proteins including integrin 2, macrophage-1 antigen, and lymphocyte function-associated antigen 1 are overexpressed on the neutrophil membrane, together facilitating the transmigration

The antiapoptotic effect of DHEA is dose dependent with an EC50 at nanomolar concentrations (EC 50 : 11.23.6 nM and 12.42.2 nM in DU145 and Caco2 cells, respectively) [1]

Current researchers have identified quite a few other exogenous RTAs and many endogenous RTAs that play a role in iron-death-mediated diseases (e.g., Table 1)