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Alternatively, the increased longevity could have resulted from decreased product-inhibition of the nematode homolog of cytoplasmic GPD1 by decreasing glycerol-3-phosphate levels, leading to increased flux through GPD1 to oxidize the cytoplasmic [NAD + ]/[NADH], a known prolongevity intervention ( 2 S-generating compound ergothioneine was shown to stimulate GPD1 and G3PS activity to extend longevity in C

Furthermore, recent studies suggested that GSH oral administration in liposomal or sublingual forms may have a better bioavailability, with a favorable impact on systemic GSH levels (29, 30)

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Nishiyama J, Matsuda K, Kakegawa W, Yamada N, Motohashi J, Mizushima N, et al