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A prior study (Dasari et al., 2010) revealed that 27-OH induced toxicity in RPE cells via the stimulation of A142 peptide production, elevation of stress markers specific to the endoplasmic reticulum (cysteinyl asparaginase-12 and C/EBP homologous protein), reduction in dysregulation of Ca 2+ homeostasis, mitochondrial membrane potential, oxidative stress (as indicated by glutathione depletion and reactive oxygen species (ROS) production), and apoptosis

It keeps the area clean, lowers the risk of infection, calms irritation, fosters healing, and helps the skin repair itself properly

Ketone bodies may increase UCP expression through regulating Sirtuin 1 activity (58), potentially via altered NAD+ levels (59), although the precise mechanisms are not yet known

doi: 10.1016/j.cmet.2020.11.017 88 ZhouJ.QiuX.ChenX.MaS.ChenZ.WangR.et al