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FIGURE 6 Moreover, research indicates that CD44v6 interacts with the mesenchymal epithelial transition factor receptor c-Met and inhibits apoptosis through two primary mechanisms: 1) presentation of HGF to c-Met or 2) by direct interaction of c-Met or HGF with the variant exon domain of CD44v6 (Figure 6B)

Liposomal GSH is capable to bypass de novo glutathione synthesis contributing to elevating glutathione level, improving redox homeostasis and decreasing the effect of TGF- [24, 25]

Oxidative stress in autoimmune diseases: an under dealt malice

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