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Concurrently, it induces tumor cell apoptosis and inhibits pro-inflammatory factors (TNF-/IL-6), providing a microbe-photothermal-antioxidant tri-modal strategy for the synergistic treatment of inflammation-associated tumors

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For example, vitamin C (Vit.C) reduces the production of free radicals, maintains GSH homeostasis, and induces the expression of HO-1, which is critical in limiting cellular damage by maintaining redox homeostasis within the brain ( in vivo study to test the neuroprotective action of Vit.C in terms of METH exposure has not been conducted