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Collectively, these findings demonstrate that the pharmacological induction of ferroptosis is a promising therapeutic strategy for TNBC

This secondary pathway activates cell-survival signaling cascades (such as PI3K/Akt) that help protect tissues from oxidative stress and inflammation, explaining many of GHRP-6's observed tissue-protective benefits in preclinical models including cardioprotection, neuroprotection, and anti-fibrotic effects [2][14]

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Selenium Ses primary functions are antioxidant (Reference Kieczykowska, Kocot and Padzior140) , involving enzymes that maintain redox homeostasis, a process influenced by its organic status (Reference Mehdi, Hornick and Istasse141)