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Similarly, brown adipose tissue (BAT) undergoes a fuel shift from glucose to fatty acid and amino acid metabolism during cold adaptation, facilitating thermogenesis to maintain body temperature ( 35 )

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One new hypothesis is that mitochondrial ROS generated during hypoxia promote the oxidation of cysteine residues within PHD2 resulting in oxidative PHD2 homodimerization and inactivation, leading to HIF- protein stabilization 26

Activated AMPK is able to indirectly inhibit the mechanistic target of rapamycin complex 1 (mTORC1) by counteracting the inhibitory effects of mTORC1 and IRS-1(75)