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Prospective longitudinal studies are essential to answer this

In vitro visualization of changes in lipid oxidation and labile iron in primary cells from PCBP1-deficient livers is a powerful approach to real-time monitoring of the function of the iron chaperone and intracellular redox status

A convincing answer could come from suitable site mutations in these proteins causing a decreased affinity for GSSG

If prevention fails, holistic and personalized management should be taken as soon as possible through timely detection and diagnosis, considering target organ protection, comorbidities, treatment goals, and other factors in reality