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At this level, it binds mtDNA, forming chemical adducts that hinder mtDNA replication and inhibit mtDNA transcription, thereby suppressing mitochondrial protein levels and producing misfolding proteins, which produce mitochondrial dysfunction [101]

Many studies are small, short-term or observational (not causal)

Zhao and Moore, 2018) strikingly reversed that dogma, opening the possibility to employ stem cells as new tools for therapeutic purposes in neural regeneration/repair in diseases and after injuries (Figure 6)

To exclude this possibility, we employed an anti-PDGFR specific neutralizing monoclonal antibody in the monotherapy and combination therapeutic settings