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Intracellular glutathione levels determine cerebellar granule neuron sensitivity to excitotoxic injury by kainic acid

Together, these findings demonstrate that, in contrast with acute exposure, chronic disruption of iron homeostasis with consequent GSH depletion remodels cellular redox homeostasis over time, inducing a state we term chronoferroptosis: a persistent ferroptotic adaptation characterized by coordinated alterations in iron-handling and antioxidant defense proteins that may represent early vulnerability to neurodegenerative pathology

This leads to the accumulation of unsaturated fatty acids, ultimately triggering ferroptosis in CD8 + T cells (115)

JAMA 287 , 742748 (2002)