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(2012) showed that knockout of Atg5 enhanced p53 stimulation and mitochondrial ROS formation following CP exposure

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doi: 10.1016/B978-0-443-16080-6.00004-5 29

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This work presents a design optimization process that incorporates both considerations

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On the other hand, excess ROS release (e.g., during an uncontrolled eosinophil activation in a flare) causes tissue damage and amplifies inflammation

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