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In recent decades, the following treatments for IC/BPS have been recommended as replenishment of the glycosaminoglycan layer to control urothelial defects, administration of amitriptyline or imipramine to inhibit neurological hyperactivity, suppression of allergies by antihistamines, and non-steroid anti-inflammatory drugs for pain control (Homma et al., 2020)

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However, in neoplastic cells, mutp53 has been shown to generate low levels of miR-34a, which enables PD-L1 overexpression (Cortez et al., 2016)