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The study began with the stimulation of murine bone marrow-derived macrophages (BMDMs) with TLR ligands under conditions of carbon starvation which comprised no glucose, pyruvate or glutamine

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Rapamycin-induced autophagy ameliorates GSH depletion-induced premature cell senescence It was shown that autophagy activation especially in the presence of an increasing autophagic flux inhibits the senescence of RPE cells 35,36,37,38,39

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