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As discussed earlier, gut microbiota dysbiosis exacerbates neuroinflammation in PND through the gut-brain axis

From a mechanistic perspective, peroxisomal -oxidation dysfunction in AP would be expected to exert a dual detrimental effect: first, by impairing the shortening of fatty acids before their transfer to mitochondria, and second, by weakening peroxisomal control of hydrogen peroxide and related redox signals