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In various tumor types, p53 mutations not only result in the loss of its tumor suppressor function but may also confer new oncogenic functions to the mutated p53, a phenomenon termed gain-of-function (GOF) ( The p53 protein plays a crucial role in cell cycle regulation and apoptosis

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Among these factors, c-Myc exhibited the most significant differential expression, and subsequent experimental validation further confirmed that c-Myc was markedly downregulated under Gln deprivation (Fig

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