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Recent studies have demonstrated that mitochondrial iron overload in FRDA also activates ferroptosis, a regulated cell death pathway driven by iron-dependent lipid peroxidation, providing a mechanistic link between iron dyshomeostasis and neuronal loss [125]

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10.3748/wjg.v20.i23.7260 41 FarcasM.GavreaA

[9] Intracellular A Juxtamembrane segment that contains: a serine residue (Ser 985), which inhibits the receptor kinase activity upon phosphorylation [10] a tyrosine (Tyr 1003), which is responsible for MET polyubiquitination, endocytosis, and degradation upon interaction with the ubiquitin ligase CBL [11] Tyrosine kinase domain, which mediates MET biological activity