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These multifaceted bioactivities significantly highlight ALAs therapeutic potential, particularly in counteracting oxidative stress and inflammation-related metabolic deficiency (20)

Structural basis of death domain interactions of Fas and FADD

FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in mice

For instance, individuals with the AS3MT rs9527 variant are considered slow methylators, which results in elevated monomethylarsonic acid (MMA) levels and heightened risk of systemic toxicity [80, 81]