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In an attempt to unify our apparently contradictory in-vitro observations with respect to findings made in vivo, we proposed the following hypothesis

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Nonetheless, as we shall see, even when tinnitus is triggered by repeated loud noise exposure, ototoxic drugs, some age-related phenomena, or inherited disorders, the subcellular alterations recapitulate those occurring during degenerative tinnitus, and there is a remarkable overlapping between the molecular pathways being affected