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doi: 10.1212/WNL.30.7.772

We assume that CSF-cNs suffer from iron accumulation, and the autophagy flux changed, and maybe an important mechanism of cognitive dysfunction after mTBI

Research into the pathways and interactive networks associated with ferroptosis in various pathological as well as physiological circumstances will greatly advance our comprehension of ferroptosis and how it might be beneficial in in treating human diseases

In addition to activating NRF2 and inducing antioxidant enzymes, some NRF2 activators may also affect other signaling pathways, potentially interfering with related biological processes